ureteral neuromuscular dysplasia - ترجمة إلى العربية
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ureteral neuromuscular dysplasia - ترجمة إلى العربية

SMOOTH MUSCLE RELAXANT ARE THE DRUGS THAT REDUCES THE TONE OF SMOOTH MUSCLES
Paralytic agent; Neuromuscular blocker; Neuromuscular blocking drugs; Neuromuscular blocking drug; Quaternary ammonium muscle relaxants; Neuroblocker; Neuromuscular blocking agent; Neuromuscular depolarizing agent; Neuromuscular nondepolarizing agent; Non-depolarizing blocking agent; Depolarizing blocking agent; Non-depolarising muscle relaxant; Neuromuscular-blocking drugs; Depolarizing neuromuscular blocking agent; Neuroparalyzer; Neuromuscular blockade; Motor block; Train of four; Paralytic agents; Neuromuscular-blocking agent; Neuromuscular-blocker; Non-depolarizing; Neuromuscular blocking medication; Neuromuscular blockers
  • Fig.1 A simple illustration of how two [[acetylcholine]] molecules bind to its receptive sites on the nicotinic receptor
  • Fig.2 A simple illustration of how [[decamethonium]] binds to the nicotinic receptor. The onium heads bind to two separate subunits of the ion-channel
  • [[Mind Map]] showing a summary of '''Neuromuscular depolarizing agent'''
  • [[Mind Map]] showing a summary of '''Neuromuscular nondepolarizing agent'''
  • [[Myofibril]]}}
  • [[Mitochondrion]]}}

ureteral neuromuscular dysplasia      
‎ خَلَلُ التَّنَسُّجِ الحَالِبِيُّ العَصَبِيُّ العَضَلِيّ‎
neuromuscular blocker         
‎ مُحْصِرٌ عَصَبِيٌّ عَضَلِيّ‎
motor end-plate         
  • 3=[[Ligand]] (such as [[acetylcholine]])}} When ligands bind to the receptor, the [[ion channel]] portion of the receptor opens, allowing ions to pass across the [[cell membrane]].
  • thumb
  • At the neuromuscular junction, the nerve fiber is able to transmit a signal to the muscle fiber by releasing ACh (and other substances), causing muscle contraction.
  • Muscles will contract or relax when they receive signals from the nervous system. The neuromuscular junction is the site of the signal exchange. The steps of this process in vertebrates occur as follows:(1) The action potential reaches the axon terminal. (2) Voltage-dependent calcium gates open, allowing calcium to enter the axon terminal. (3) Neurotransmitter vesicles fuse with the presynaptic membrane and ACh is released into the synaptic cleft via exocytosis. (4) ACh binds to postsynaptic receptors on the sarcolemma. (5) This binding causes ion channels to open and allows sodium and other cations to flow across the membrane into the muscle cell. (6) The flow of sodium ions across the membrane into and potassium ions out of the muscle cell generates an action potential which travels to the myofibril and results in muscle contraction.Labels:A: Motor Neuron AxonB: Axon TerminalC. Synaptic CleftD. Muscle CellE. Part of a Myofibril
  • Motor Endplate
JUNCTION BETWEEN THE AXON OF A MOTOR NEURON AND A MUSCLE FIBER
Nueromuscular junction; Motor end plate; Motor end-plate; Neuromuscular junctions; Neuromuscular; Motor endplate; Neuromuscular Junction; Neuromuscular junction diseases; Neuromuscular agents; Myoneural junction; Myoneural; Neuromuscular plate; Neuromuscular transmission; End plate; Skeletal muscle junction; Neuromuscular block; Motor end organ; Nerve-Muscle Interface; Muscular block; Bromage score; Neuromuscular system; Neuromuscular synapse
صَفيحَةٌ انْتِهائِيَّةٌ حَرَكِيَّة

تعريف

neuromuscular
¦ adjective relating to nerves and muscles.

ويكيبيديا

Neuromuscular-blocking drug

Neuromuscular-blocking drugs block neuromuscular transmission at the neuromuscular junction, causing paralysis of the affected skeletal muscles. This is accomplished via their action on the post-synaptic acetylcholine (Nm) receptors.

In clinical use, neuromuscular block is used adjunctively to anesthesia to produce paralysis, firstly to paralyze the vocal cords, and permit intubation of the trachea, and secondly to optimize the surgical field by inhibiting spontaneous ventilation, and causing relaxation of skeletal muscles. Because the appropriate dose of neuromuscular-blocking drug may paralyze muscles required for breathing (i.e., the diaphragm), mechanical ventilation should be available to maintain adequate respiration.

Patients are still aware of pain even after full conduction block has occurred; hence, general anesthetics and/or analgesics must also be given to prevent anesthesia awareness.